2014-2016 | bislab

2016

ERK-mediated phosphorylation of BIS regulates nuclear translocation of HSF1 under oxidative stress. Kim HY et al. Exp Mol Med 2016.84, e260 (IF 5.06) Link
BIS-mediated STAT3 stabilization regulates glioblastoma stem cell-like phenotypes. Im CN et al. Oncotarget 2016:7 (23), 35056-35070 (IF 5.17) Link
Myosin heavy chain is stabilized by BCL-2 interacting cell death suppressor (BIS) in skeletal muscle. Hong J et al., Exp Mol Med 2016:48, e255 (IF 5.06) Link
Depletion of BIS sensitizes A549 cells to treatment with cisplatin. Cui MN et al., Mol Cell Toxicol 2016:12(1), 63-71 (IF 1.25) Link
Hypertrophic cardiomyopathy due to mitochondrial DNA mutation diagnosed by pathology and gene sequencing. Choi Y et al., Circulation 2016:133, 1297-1299 (co-work)
In vivo near-infrared imaging for the tracking of systemic delivered mesenchymal stem cells: tropism for brain tumor and biodistribution. Kim SM et al., Int J Nanomed 2016:11, 13-23 (co-work)

2015

Enhancement of SOX-2 expression and ROS accumulation by cultivation of A172 glioblastoma cells under non-adherent culture conditions. Im CN et al., Oncol Rep 2015: 34(2), 920-928 (IF 2.66) Link
Bis Expression in Patients with Surgically Resected Lung Cancer and its Clinical Significance.Yeo CD et al., Ann Surg Oncol 2015 :22, S1365-1370 (IF 4.04) Link

2014

Bis Targeting induces Cellular Senescence through regulation of 14-3-3 Zeta/ STAT3/ Skp2/p27 in Glioblastoma Cells. Lee JJ et al., Cell Death Dis 2014:20, e1537 (IF 5.97) Link
Bis is induced by oxidative stress via activation of HSF1. Yoo HJ and Im CN et al., Korean J Physiol Pharmaco 2014:18, 403-409 (IF 2.06) Link
Aggravation of diabetic nephropathy in BCL-2-interacting cell death suppressor (Bis) haploinsufficient mice together with impaired induction of superoxide dismutase (SOD) activity. Lim JH and Youn DY et al., Diabetologia 2014:57, 214-223 (IF 6.08) Link
Downmodulation of Bis reduces invasive ability of glioma cells induced by TPA through NF-κB mediated MMP-9 activation. Lee YD et al., BMB Rep 2014: 47, 262-267 (IF 3.09) Link